Ocular burns constitute a true ophthalmic emergency and are a major cause of preventable visual impairment worldwide. Injury most commonly results from chemical exposure, particularly alkali agents, but may also follow acid, thermal, or radiation exposure. Risk factors include occupational hazards, industrial accidents, household chemical access, and inadequate eye protection. Tissue damage is mediated by direct cytotoxicity, inflammation, ischemia, and disruption of limbal stem cell function, with alkali injuries producing rapid and deep ocular penetration. Clinical presentation ranges from conjunctival hyperemia and epithelial defects to corneal opacification, elevated intraocular pressure, and extensive limbal ischemia. Diagnosis is clinical and relies on prompt assessment of ocular surface integrity, limbal involvement, and anterior segment damage. Management prioritizes immediate and copious irrigation, followed by targeted medical and surgical interventions based on injury severity. Complications include corneal scarring, limbal stem cell deficiency, glaucoma, chronic inflammation, and dry eye disease. Visual prognosis depends on injury mechanism, extent of limbal involvement, and timeliness of intervention. This activity for healthcare professionals is designed to enhance learners’ competence in evaluating and managing ocular burns. Participants will advance their mastery of the condition’s etiology, risk factors, pathophysiology, clinical presentation, and evidence-based diagnostic and therapeutic approaches. Improved skills will equip clinicians to collaborate with interprofessional teams providing care for affected individuals.
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- Article / monograph / reference
- Credit · Up to 2.5 credits
- MOC · Up to 2.5 points
- Fee varies · this provider typically $49–98
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ReleasedSep 1, 2026
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Last day to claim creditSep 1, 2029 3 years left
