Conduction aphasia is a relatively rare form of fluent aphasia characterized by the inability to repeat sentences, defective use of phonemes, and impaired naming ability. By far, the most common antecedent of conduction aphasia is a cerebrovascular event (stroke). According to the classical categorization of aphasic syndromes, frontal lesions cause motor aphasias (impaired language production,) and temporal parietal lesions produce sensory aphasias (impaired language comprehension), while lesions connecting the frontal and temporal lobes through the white matter tract known as the arcuate fasciculus cause conduction aphasia. Language production and auditory comprehension remain relatively preserved in conduction aphasia, distinguishing it from the more familiar non-fluent (Broca’s) and semantic (Wernicke’s) aphasias. This activity reviews the underlying etiology and pathophysiology of conduction aphasia, examines when conduction aphasia should be considered in the differential diagnosis of aphasia, and clarifies how to evaluate conduction aphasia properly. This activity provides healthcare professionals the knowledge and tools to improve patient care for this complex condition.
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